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Clinician Info

Short Term Effects

  • Production and release of beta-endorphins (these are morphine like substances produced by various cells in the body that inhibit the sensation of pain).
  • Cortisol production is increased (cortisol is the precursor of cortisone). This enables the body to combat the stress associated with trauma or the disease process.
  • The short-term effect is significant in 5-10% of cases during or after the conclusion of the initial treatment, but is not as important as the long-term or cumulative effect.

Long Term or Cumulative Effects

  • ATP (adenosine triphosphate) production is increased resulting in improved cellular metabolism.
  • DNA (deoxyribonucleic acid) production, the protein building block of tissue is substantially increased.
  • Neurotransmission is facilitated due to elevated levels of serotonin and acetylecholine.
  • Mitochondrial activity is stimulated resulting in cell replication etc.
  • Modulation of macrophages, fibroblasts and other cells.
  • Angiogenesis (formation new blood vessels).
  • Regulates cell membrane potential, essential in NA, CL and K ion transfer (electrolyte balance).
  • Cytokines and other chemicals enhancing cellular communications are released.

Other Effects

  • The immune response is stimulated.
  • Lymphatic drainage is improved.
  • The histamine response is positively altered.
  • Production of growth hormone is increased.
  • Stimulation of the healing processes is accompanied by relief of symptoms.

It should be noted that many other positive physiological activities are modulated and extensive research is currently in progress to fully explore these changes.